A new in vitro method for identifying chemical sensitizers combining peptide binding with ARE/EpRE-mediated gene expression in human skin cells.

Article Details

Citation

McKim JM Jr, Keller DJ 3rd, Gorski JR

A new in vitro method for identifying chemical sensitizers combining peptide binding with ARE/EpRE-mediated gene expression in human skin cells.

Cutan Ocul Toxicol. 2010 Sep;29(3):171-92. doi: 10.3109/15569527.2010.483869.

PubMed ID
20491607 [ View in PubMed
]
Abstract

Allergic contact dermatitis (ACD) is a significant safety concern for developers of cosmetic, personal care, chemical, pharmaceutical, and medical device products. The guinea pig maximization test (GMPT) and the murine local lymph node assay (LLNA) are accepted methods for determining chemical sensitization. Recent legislative initiatives in Europe require the development of new in vitro alternatives to animal tests for chemical sensitization. The aim of this project was to develop an in vitro screening method that uses a human skin cell line (HaCaT), chemical reactivity, and gene expression profiling to identify positive and negative responses, to place chemicals into potency categories of extreme/strong (ES), moderate (M), weak (W), and nonsensitizers (N), and to provide an estimate of corresponding LLNA values. The method and processing algorithm were developed from a training set of 39 chemicals possessing a wide range of sensitization potencies. Three cationic metals, chromium (Cr), nickel (Ni), and silver (Ag), were also evaluated in this model. Chemical reactivity was determined by measuring glutathione (GSH) depletion in a cell free matrix. Three signaling pathways (Keap1/Nrf 2/ARE/EpRE, ARNT/AhR/XRE, and Nrf1/MTF/MRE) that are known to be activated by sensitizing agents were monitored by measuring the relative abundance of 11 genes whose expression is controlled by one of these 3 pathways. Final exposure concentrations were based on toxicity and solubility. A range-finding experiment was conducted with each compound to determine cytotoxicity and solubility. Six exposure concentrations (0.1 to 2,500 microM) and an exposure time of 24 hours were used in the final experiments. Glutathione depletion alone did not provide the accuracy necessary to differentiate potency categories. However, chemical reactivity combined with gene expression profiles significantly improved the in vitro predictions. A predicted toxicity index (PTI) was determined for each test chemical. A comparison of LLNA values with PTI values revealed an inverse relationship. The large variation in LLNA data for compounds in the same potency category makes direct extrapolation from PTI to LLNA difficult. To challenge the system, 58 additional compounds were submitted in a blinded manner. Compounds placed into ES and M categories were considered positive, whereas compounds classified as W or N were considered negative. Accuracy was approximately 84%, with a sensitivity of 81% and a specificity of 92%. The model correctly identified 2 of 3 cationic metals as positive. In conclusion, the method described here demonstrates a valuable in vitro method for identifying chemicals and metals that induce skin sensitization.

DrugBank Data that Cites this Article

Pharmaco-transcriptomics
DrugDrug GroupsGeneGene IDChangeInteractionChromosome
2-AminophenolExperimentalAKR1C21646
upregulated
2-aminophenol results in increased expression of AKR1C2 mRNA10p15.1
2-AminophenolExperimentalCXCL83576
upregulated
2-aminophenol results in increased expression of CXCL8 mRNA4q13.3
2-AminophenolExperimentalCYP1A11543
upregulated
2-aminophenol results in increased expression of CYP1A1 mRNA15q24.1
2-AminophenolExperimentalHMOX13162
upregulated
2-aminophenol results in increased expression of HMOX1 mRNA22q12.3
2-AminophenolExperimentalNQO11728
upregulated
2-aminophenol results in increased expression of NQO1 mRNA16q22.1
Benzoic acidApproved InvestigationalMT1A4489
upregulated
Benzoic Acid results in increased expression of MT1A mRNA16q13
Benzoic acidApproved InvestigationalMT2A4502
upregulated
Benzoic Acid results in increased expression of MT2A mRNA16q13
Benzoic acidApproved InvestigationalTXN7295
upregulated
Benzoic Acid results in increased expression of TXN mRNA9q31.3
2-mercaptobenzothiazoleApproved Experimental Vet ApprovedAKR1C21646
upregulated
captax results in increased expression of AKR1C2 mRNA10p15.1
2-mercaptobenzothiazoleApproved Experimental Vet ApprovedCYP1A11543
upregulated
captax results in increased expression of CYP1A1 mRNA15q24.1
2-mercaptobenzothiazoleApproved Experimental Vet ApprovedGCLC2729
upregulated
captax results in increased expression of GCLC mRNA6p12.1
DinitrochlorobenzeneInvestigationalAKR1C21646
upregulated
Dinitrochlorobenzene results in increased expression of AKR1C2 mRNA10p15.1
DinitrochlorobenzeneInvestigationalCXCL83576
upregulated
Dinitrochlorobenzene results in increased expression of CXCL8 mRNA4q13.3
DinitrochlorobenzeneInvestigationalCYP1A11543
upregulated
Dinitrochlorobenzene results in increased expression of CYP1A1 mRNA15q24.1
DinitrochlorobenzeneInvestigationalMAFF23764
upregulated
Dinitrochlorobenzene results in increased expression of MAFF mRNA22q13.1
DinitrochlorobenzeneInvestigationalMT2A4502
upregulated
Dinitrochlorobenzene results in increased expression of MT2A mRNA16q13
DinitrochlorobenzeneInvestigationalNQO11728
upregulated
Dinitrochlorobenzene results in increased expression of NQO1 mRNA16q22.1
DiphencyproneInvestigationalHMOX13162
upregulated
diphenylcyclopropenone results in increased expression of HMOX1 mRNA22q12.3
DiphencyproneInvestigationalMAFF23764
upregulated
diphenylcyclopropenone results in increased expression of MAFF mRNA22q13.1
DiphencyproneInvestigationalMT1A4489
upregulated
diphenylcyclopropenone results in increased expression of MT1A mRNA16q13
DiphencyproneInvestigationalMT2A4502
upregulated
diphenylcyclopropenone results in increased expression of MT2A mRNA16q13
DiphencyproneInvestigationalTXN7295
upregulated
diphenylcyclopropenone results in increased expression of TXN mRNA9q31.3
FormaldehydeApproved Vet ApprovedAKR1C21646
upregulated
Formaldehyde results in increased expression of AKR1C2 mRNA10p15.1
FormaldehydeApproved Vet ApprovedCYP1A11543
upregulated
Formaldehyde results in increased expression of CYP1A1 mRNA15q24.1
FormaldehydeApproved Vet ApprovedGCLC2729
upregulated
Formaldehyde results in increased expression of GCLC mRNA6p12.1
FormaldehydeApproved Vet ApprovedTXN7295
upregulated
Formaldehyde results in increased expression of TXN mRNA9q31.3
GlycerinApproved InvestigationalHMOX13162
upregulated
Glycerol results in increased expression of HMOX1 mRNA22q12.3
GlycerinApproved InvestigationalMT1A4489
upregulated
Glycerol results in increased expression of MT1A mRNA16q13
GlycerinApproved InvestigationalMT2A4502
upregulated
Glycerol results in increased expression of MT2A mRNA16q13
PhenylacetaldehydeExperimentalAKR1C21646
upregulated
phenylacetaldehyde results in increased expression of AKR1C2 mRNA10p15.1
Propyl GallateInvestigationalAKR1C21646
upregulated
Propyl Gallate results in increased expression of AKR1C2 mRNA10p15.1
Propyl GallateInvestigationalCYP1A11543
upregulated
Propyl Gallate results in increased expression of CYP1A1 mRNA15q24.1
Propyl GallateInvestigationalHMOX13162
upregulated
Propyl Gallate results in increased expression of HMOX1 mRNA22q12.3
Propyl GallateInvestigationalMAFF23764
upregulated
Propyl Gallate results in increased expression of MAFF mRNA22q13.1
Salicylic acidApproved Investigational Vet ApprovedAKR1C21646
upregulated
Salicylic Acid results in increased expression of AKR1C2 mRNA10p15.1
Salicylic acidApproved Investigational Vet ApprovedMAFF23764
upregulated
Salicylic Acid results in increased expression of MAFF mRNA22q13.1
Salicylic acidApproved Investigational Vet ApprovedMT1A4489
upregulated
Salicylic Acid results in increased expression of MT1A mRNA16q13
Salicylic acidApproved Investigational Vet ApprovedTXN7295
upregulated
Salicylic Acid results in increased expression of TXN mRNA9q31.3
Silver nitrateApproved InvestigationalCXCL83576
upregulated
Silver Nitrate results in increased expression of CXCL8 mRNA4q13.3
Silver nitrateApproved InvestigationalMT2A4502
upregulated
Silver Nitrate results in increased expression of MT2A mRNA16q13
Sodium lauryl sulfateExperimentalAKR1C21646
upregulated
Sodium Dodecyl Sulfate results in increased expression of AKR1C2 mRNA10p15.1
Sodium lauryl sulfateExperimentalCXCL83576
upregulated
Sodium Dodecyl Sulfate results in increased expression of CXCL8 mRNA4q13.3
Sodium lauryl sulfateExperimentalCYP1A11543
upregulated
Sodium Dodecyl Sulfate results in increased expression of CYP1A1 mRNA15q24.1
Sodium lauryl sulfateExperimentalMAFF23764
upregulated
Sodium Dodecyl Sulfate results in increased expression of MAFF mRNA22q13.1
Sodium lauryl sulfateExperimentalMT1A4489
upregulated
Sodium Dodecyl Sulfate results in increased expression of MT1A mRNA16q13
Sodium lauryl sulfateExperimentalNQO11728
upregulated
Sodium Dodecyl Sulfate results in increased expression of NQO1 mRNA16q22.1
Sodium lauryl sulfateExperimentalTXN7295
upregulated
Sodium Dodecyl Sulfate results in increased expression of TXN mRNA9q31.3